How Do We Get Acne

Acne is a chronic inflammator disease sebaceous gland and hair follicle.
Dissemination of the lesions (injuries) caused by acne, it can show as an individual
or the common nodes, called modules, population (acne, mitiseri).
Lesions are primarily associated with sebaceous gland that surrounds the hair follicle in the skin, it is visible fat. Lesions are most often planted in the face, neck, chin, wattles, shoulders, the rear of the humeri, back.

Acne in black or white top of the result of abnormal flow sebuma (fats --
tallow). Both the type is reflected in various forms: nodes, modules, cysts, postule. White Peaks are keratonostic nodes, which fill overflow channels glands, black top is also keratonostic plug gaps in the glands, but not completely blocked glands.

The formation of acne is associated with a range of factors. First, the increased production sebuma, which blocks the hair follicle. In the lock of hair follicle is the normal population of the bacteria dramatically increased (reproduced). The bacteria produce (secrete) inflamatory chemicals that penetrate the skin and cause inflammation.
Normal operations against acne include:
- Reduction (decrease) in inflammatory diseases of follicle,
- The restriction of bacterial growth.

Acne is very common in young people in adolescence. Most occur in the early
adolescence and the decline after 25 year. Some people, mostly women, receive
Acne first time in my life in the late 20th or 30 years.

Also, the impact of heredity in the formation of acne, more often in men.
Acne may also occur in individuals with endocrine changes, in
example, abnormal hormone Circulation. Also, different drugs may stimulate and
worsen the condition of acne.